A 40-year-old Indian female patient presented with hirsutism. She had excess hair growth, especially on her face since 2015. Examination in 2015 did not show an endocrine disorder and she started laser therapy on a regular basis. Despite laser therapy, excess facial hair increased. She didn’t have any other skin abnormalities, nor alopecia androgenetica. Her voice was not deepened and her muscle mass was not increased. The menstrual cycle was regular. She did not use any medication, except for etanercept for rheumatoid arthritis. She did not smoke or use alcohol. On physical examination she had excess hair distributed equally all over her body, except for her face due to laser therapy. However, a picture from 2016, before laser therapy, showed excess facial hair, especially on her cheeks and temples. Importantly, the excess hair did not have a male-pattern, and therefore hirsutism was excluded. Hypertrichosis, with predominantly excess hair growth on cheeks and temples has a different differential diagnosis as compared to hirsutism. Laboratory examination showed normal renal- and thyroid function. Erythrocyte sedimentation rate (43 mm/h), ferritin(279 g/L) and alanine transaminase (67 IU/L) were slightly increased. Hepatitis C-, HIV-, and SLE serology, and HFE mutation analysis were negative. X-thorax and abdominal ultrasound did not show a malignancy. Urine porphyrine test however, demonstrated Porphyria Cutanea Tarda (PCT), with increased uro- (173 nmol/mmol), hepta- (113 nmol/mmol), hexa- (12 nmol/mmol), and pentaporphyrine (16 nmol/mmol) creatinine ratios. PCT is mostly sporadic and is caused by diminished activity of hepatic uroporphyrinogen decarboxylase. Although blistering of sun-exposed skin is the most common feature in PCT, PCT may present with solely hypertrichosis over the face and upper torso, as in our patient. Recently, flebotomy was started, resulting in decreased uroporphyrine levels. Few months after normalizing porphyrine levels, hypertrichosis can be expected to gradually disappear.